Sentence examples for intestinal resistance from inspiring English sources

Annexins contribute to the intestinal resistance to injury, as they possess anti-inflammatory properties as well as gastroprotective properties.

However, there is little evidence that these non-digestible carbohydrates strengthen intestinal resistance to infection and gut barrier function.

However, both in normotensive and in hemorrhagic cats, the renin-angiotensin and vasopressin systems have been reported to be redundant mechanisms with respect to intestinal vasoconstriction, as in the absence of one control system the other maintains intestinal resistance [ 29].

In addition, defensins, which are antimicrobial peptides secreted by human and rodent ileal Paneth cells, contain 6 conserved cysteines[ 37] and are important for intestinal resistance to Salmonella infection[ 38].

In addition, the induction of conserved innate immune pathways (such as the Imd and STAT pathways) and the production of AMPs and ROS as basic components of the intestinal resistance to infection support the utility of this model for examining basic mechanisms of mammalian intestinal epithelial homeostasis, regeneration and disease.

This study shows that insulin is a potent stimulator of GU in the healthy intestine and that intestinal insulin resistance is ameliorated after bariatric surgery.

Intestinal colonization resistance is defined as the resistance to colonization by ingested bacteria or inhibition of overgrowth of resident bacteria normally present at low levels within the intestinal tract [ 7].

In our data, obese individuals had intestinal insulin resistance regardless of their glycaemic status, suggesting that intestinal insulin resistance may occur before, and independently of, systemic insulin resistance.

To evaluate the cellular effects of Sb on intestinal epithelium resistance, we infected human T84 cell monolayer by ST. Results in figure 4A show that ST infection diminished gradually the electrical resistance (TER) of monolayers.

In conclusion, this study shows that intestinal insulin resistance is ameliorated after bariatric surgery.

In our study, obese individuals had intestinal insulin resistance regardless of their glycaemic status.