The interface cross-section microstructure of such adhesion jointing surfaces is discussed.
A strength-based criterion is monitored in each ply using a local model of the skin stiffener interface cross-section.
For the numerical analysis, two-dimensional models of the interface cross-section were used with a strength-based criterion that monitored failure within each ply.
To study the interface, AFM phase image were obtained from interface (cross-section) of 30 nm thin Pd film deposited over STO perovskite substrate, shown in Fig. 4. The area in the left of the Pd film (shiny line) corresponds to substrate, and the right part of this line is artifact due to tip dimension.
Cellular proliferation at the meniscal tissue surface, surface interface, cross-section, and cross-section interface were decreased by IL-1 in both inner and outer meniscal repair explants.
Cellular proliferation at the meniscal tissue surface, surface interface, cross-section, and cross-section interface were decreased in the presence of TNF-α in both inner and outer meniscal repair explants.
In both the inner and outer meniscal repair explants, TGF-β1 treatment did not appear to alter cellular proliferation at the meniscal tissue surface, surface interface, cross-section, or cross-section interface.
The cross section interface included a 50 μm region on either side of the interface, inclusive of the interface, for each of these three layers.
Open image in new window Fig. 6 Histology showing bone implant interface in rat tibia cross section, 63 days after the implantation.
Gurmani et al. ( 2011) simulated the same system with approximately 2-nm length sides for the cross section of the interface.
The interfacial bond strength, i.e., the bond between a coating and its substrate (steel), was determined by performing microindentation tests, on a cross section near the interface.
