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The discrete transition of the fibre orientation at the media adventitia interface can be explained by accounting for the different reference configurations of both layers.
Further, the large variety of identified protein complexes in the GRN/PPN interface can be explained by the vital spatial and temporal dependency of genes that belong to the same protein complex to be functional.
This dichotomy, where microcapsule formation is not only possible, but occurs only when a specific copolymer from the pair is drawn to the interface can be explained through the supramolecular assembly process.
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This discrepancy with our data showing that pure zwitterionic interfaces bind Penetratin can be explained taking into account that in some of the previous studies, the authors used Small Unilamellar Vesicles (SUV) with a highly positive curvature of membranes.
The dependence of hole mobility on well width can be explained by an interface roughness of 0.4 nm, in accordance with the X-ray reflectivity measurements, and the associated fluctuation of the strain at the interfaces.
It is shown that strained devices present higher noise than conventional ones, independent on the fin width, which can be explained by poorer interface quality observed in strained devices.
This observation can be explained by the different interfaces used for interaction.
This can be explained by the fact that interfaces in this category are particularly important for care in which mistakes are potentially disastrous, such as in medication care [ 17].
These changes can be explained by the decrease in the interface effect due to the introduction of the weak oceanic crust.
This can be explained by the higher energy consumption in the LR interface (IEEE802.11g) compared to the SR interface (WiMedia), as well as the higher data rates achieved on the SR links.
The more apparent hypo-biofilm phenotypes of mutant LapD variants DK and YR can be explained by disturbance of the LapG binding interface by these mutations.
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