Sentence examples for interactions of these pathways from inspiring English sources

Although it has been demonstrated that disruption of either of these pathways has a profound effect on T-cell development, we do not yet have an understanding of the dynamical interactions of these pathways in their joint shaping of the T cell repertoire.

However, due to the complex interactions of these pathways, treatments that target only one protein may not be very effective.

Further studies are necessary to determine if there are comparable interactions of these pathways with, respectively, ionotropic and metabotropic receptors as has been suggested [15], [65].

In future studies, the interactions of these pathways will be explored in detail.

A model was proposed to show the possible roles and interactions of these pathways for the sterile gynoecium development.

Second, although the biochemical interactions of these pathways are quite complex, there is quite a bit of knowledge of how these pathways function.

Increasing our understanding for the pathways involved in hypertrophy and atrophy and particularly the interaction of these pathways is essential in designing therapeutic strategies for both prevention and treatment of muscle wasting conditions with age and with disease.

We generated hypomorphic mouse models of cartilage-specific loss of BMP and TGFβ signaling to assess the interaction of these pathways in postnatal growth plate homeostasis.

An understanding of the interaction of these pathways is an important avenue for developing treatments and therapies for demyelinating events.

Whether the interaction of these pathways is positive or negative and the timing of their interactions are open areas of investigation that will have an important impact on our understanding of CNS myelination.

Increasing evidence shows that there is a crosstalk between TGF β/BMP antigrowth signalling and Ras/MAP-K progrowth signalling, and it is likely that the interaction of these pathways is cell type and tissue specific.