There was a significant effect of the diet-stress interaction on body weight gain (F 2,48) = 3.46, p < 0.05).
There was a main effect of diet and interaction on body weight gain, where the post-hoc test showed that rats subjected to restraint stress from both control and ω-3 diet groups had significantly decreased body weight gain compared to the respective unstressed controls (F 1,48) = 1559, p < 0.0001).
Specifically in these analyses, we regressed the additive and dominance index values, WT, and the interactions of body weight with the index values on distance, duration, and speed.
In the present study, we found significant interactions between LU and ATO treatments on body weight, liver index, ALT, AST, T-AOC levels, GSH, and MDA contents.
Two-way ANOVA revealed no significant interaction between diet and exercise on body weight but very significant main effects for diet (F (1,52) = 6.85, p<0.01) and exercise (F (1,52) = 9.5, p = 0.01), indicating that these factors acted independently.
Two way repeated measures ANOVA was used to determine the effect of diabetes, MVT, and the interaction of these two factors on body weight, fasting blood glucose and glucose levels during oral glucose tolerance test in the entire population as well as in males or females, respectively.
Another example is the effect on body weight of the interaction between smoking tobacco and genotype (Freathy et al., 2011).
We report effects of these EDCs on body weight, sociability, social status, and interactions between these factors.
The diet*genotype interaction trend suggests that part of the effect of quinine on body weight and fat mass may be Trpm5-dependent.
We found an interaction between treatment and time [ F 6,98)=5.94, P<0.0001], indicating that the treatment did not have the same effect on body weight loss at all time points.
Diet or exercise produced significant independent effects on body weight (diet: F 1,52) = 6.85, p = 0.012; exercise: F 1,52) = 9.52, p<0.01) with no significant interaction.
