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We found no significant interaction of the polymorphisms studied here with ever use of swimming pools.
No other significant interaction of the polymorphisms was observed on the NEO PI-R or STAI factors.
It has been reported that the polymorphisms might be in linkage disequilibrium and interaction of the polymorphisms within haplotypes can be a major determinant of disease susceptibility than the individual polymorphism [ 11].
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Moreover, the interaction of the polymorphism with retinol intake on type 2 diabetes risk was assessed.
Furthermore, we studied the interaction of these polymorphisms and their interaction with physical activity.
Although associated with a lower risk of colorectal cancer (22), the exact interaction of this polymorphism with DNA methylation in the colon is not known, although interactions with folate status and genomic DNA methylation in leukocytes have been reported (23, 24).
The gene expression/polymorphism model with interaction (full B model) includes the variables of the full A model as well as an indicator variable for Δ32 polymorphism and the two-way and three way interaction of the CCR5-Δ32 polymorphism with the gene expression variables.
Furthermore, we have found a significant interaction of this polymorphism with that of the serotonin transporter where, in subjects with the s allele of 5-HTTLPR having a Val/Val genotype of BDNF, there is a 70%% probability of lithium non-response [ 59].
We observed a more-pronounced impairment in ATG16L1 KO + ATG16L1∗300A cells, suggesting functional interaction of the T300A polymorphism and reduced CLEC12A expression.
CNDP1 polymorphism was not associated with mortality, and nor was there an interaction of this polymorphism with DN for prediction of mortality in patients with type 1 diabetes.
Finally, no association was observed in the analysis of interaction between the polymorphisms studied and occupation.
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