Sentence examples for interaction complement from inspiring English sources

In contrast, genes up-regulated by changes of H3K9Ac and H3K9Me2 were primary distributed to cytokine-cytokine receptor interaction, complement and coagulation cascades and Jak-STAT signaling pathways (Table S5).

According to the results, several cellular pathways were significantly affected, such as TGF-beta signaling pathway, ECM-receptor interaction, complement and coagulation cascades, and cytokine-cytokine receptor interaction.

Thus, through the C5a C5aR interaction, complement appears to influence the extent of the pro-inflammatory and immunomodulatory responses triggered via TLRs.

In the present study, we found the global expressed and differentially expressed proteins participated in respective 32 and 62 pathways including MAPK, ECM-receptor interaction, complement and coagulation cascades, and focal adhesion KEGG pathways (Additional file 2: Table S1 and Table S3).

Through the pathway analysis, the significant pathways mainly concerned with cell adhesion molecules, cytokine-cytokine receptor interaction, complement and coagulation cascades, toll-like receptor signaling pathway, MAPK signaling pathway, suggesting that the host took different strategies to activate immune and inflammatory response upon H. parasuis infection.

In addition to these, other signaling components, such as ECM-receptor interaction, complement and coagulation, mammalian target of rapamycin (mTOR) signaling pathway, focal adhesion, and signaling pathways of TGF-beta, mitogen-activated protein kinase (MAPK), vascular endothelial growth factor (VEGF), and calcium were enriched to a greater extent in hOFs and hDFs than in hOF-iPSCs).

Their interaction, complemented by other factors, including visit and health system factors, results in some issues being addressed but not others, which are left for subsequent visits or left unaddressed [ 31].

Identification of the mTORC1-GRB10 interacomplementsements a known negative feedback loop in which mTORC1 activation can inhibit the PI3K pathway by S6K1-mediated phosphorylation and degradation of insulin receptor substrate-1 (IRS-1), and it fills an important gap in our understanding the underlying mechanisms by which mTORC1 inhibits PI3K-Akt signaling [49], [50].

Our observations from the genetic interactions complement the biochemical results demonstrating that RbbA is functionally an important component of the membrane bound YhjD.

Furthermore, SI and HC interactions complement each other well, suggesting that our approach might generate testable hypotheses and provide valuable experimental leads.

Classification of these proteins into networks revealed five significant functional processes that were affected by CEE treatment: blood coagulation, kallikrein-kinin system, cell adhesion-platelet-endothelium-leukocyte interactions, complement system, and ossification.