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It was suggested that non-thermal exposition to high-frequency electromagnetic fields may rather have a tumor promoter than an initiator effect [ 9], since DNA, generally, does not appear to be significantly altered or damaged by electromagnetic fields [ 11].
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To provide a clear answer to this question, the kinetics of Cu(I) disproportionation and RX activation were investigated in various experimental conditions that match the polymerization environment (different amine ligands and initiators, effect of solvent, halide ions and Cu0).
These curing effects were investigated as initial initiator and polymer concentrations were varied.
Ageing of mixed DPMPLi/t-BuOLi solutions as well as an excess of the alkoxide over the initiator have no effect on the stability of the growing chain-ends.
For most early initiators the rTMS effect predominantly occurred in the Non-predictive conditions; only two participants also showed considerable Predictive effects.
The involvement of a free-radical mechanism is indicated by the intermediates formed, and free-radical initiator and inhibitor effects on observed disappearance rates of phenols.
It is known that the concentration of initiator has important effects on the process of polymerization, particularly on the particle formation [23, 24].
Direct damage to DNA caused by environmental carcinogens and ionising radiation has been considered as a significant initiator of deleterious effects, such as mutation and cancer.
Changes in nuclear structures, that we identified earlier as an important initiator of microgravity effects [ 22], might explain the RhoA activity inhibition and changes in cell tension evoked under microgravity.
Effect of initiator composition on polymer length was also investigated using GPC.
The effect of initiator concentration, costabilizer concentration, surfactant concentration, sonication time and amplitude, and their interactions on the particle size were identified.
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