Exact(3)
Innate immune responses are initiated by pattern recognition receptors (PRRs), which recognize specific structures of microorganisms.
Innate immune responses in insects are initiated by pattern recognition receptors (PRRs) that recognize microbial cell wall components such as peptidoglycan and β 1,3-glucan (GRP).
Pathogen recognition is initiated by pattern recognition receptors that bind conserved stereotypical, rather than particular, molecular structures present in a wide spectrum of microorganisms but absent in the host [ 10], such as peptidoglycan (PGN), which is a major constituent of the cell wall of both Gram-negative and Gram-positive bacteria [ 11].
Similar(57)
Whether this limited overlap is due to early divergence of these regions initiated by patterning signals, resulting in distinct transcriptional contexts, remains an open question.
Complement activation is initiated by the pattern-recognition molecules complement component C1q, mannose-binding lectin (MBL) and ficolins (H-, L-, M-ficolin), which typically recognize antibody antigen complexes or foreign polysaccharides.
Virus-infected cells generally respond to infection by induction of an innate immune response that is initiated by several cellular pattern recognition receptors (PRRs), which detect specialized pathogen-associated molecular pattern (PAMPs) molecules.
It is shown in this paper that a continuous-time dynamical neural network model, employing a radial basis function and a sigmoid multi-layer perceptron sub-networks, is capable of maximizing such an energy form locally, thus performing almost perfectly nearest neighbor classification, when initiated by a distorted pattern.
The UNIPEN project [15] of data exchange was initiated by International Association of pattern recognition (TC-11) in 1992 with the objective of proposing a uniform format for representation and exchange of online data.
According to the danger model [ 37], an inflammatory response is initiated by conserved molecular patterns that may be associated with both foreign antigens and cellular components released by damaged cells.
This form of sterile inflammation is prototypically initiated by engagement of innate pattern recognition receptors, like toll-like receptors [ 68].
This pathway is initiated by bacterial stimulation of pattern recognition receptors on monocytes and macrophages, resulting in the production of cytokines that activate lymphocytes and induce IFN-γ production.
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