Sentence examples for initial phases of disease from inspiring English sources

The previous laboratory records were searched for white blood cell count (WBC), polymorphonuclear (PMN) and lymphocyte (Lymph) absolute count, hemoglobin (Hgb), platelet count (PLT), erythrocyte sedimentation rate (ESR), and C-reactive protein (CRP) in the initial phases of disease.

Services that are excluded - usually in the initial phases of disease - are provided by networks funded by local authorities [ 3], which promotes conflict between health authorities and insurance companies to avoid assuming the funding.

In the initial phase of disease, mice developed a specific Th1-driven immune response independent of inoculum concentration.

This raises the possibility that a smaller subgroup of patients has an accelerated decline rate in the initial phase of disease.

Acutely affected animals were those that survived the initial phase of disease but showed evident clinical signs (dyspnoea and cough, fever, reduced appetite, and were reluctant to move).

In 10%to15%5% of patients, the initial phase of disease is followed by more severe pulmonary disease characterized by respiratory distress, pulmonary infiltration of mononuclear inflammatory cells, multinucleated syncytia, and fibrosis (3, 4).

This result suggests that T cell recognition of HOK264 in B10.Q mice tends to correspond to manifestation of clinical signs of arthritis while K264-specific T cells could play a role in the initial phase of disease.

At the initial phase of disease (day 15 postimmunization) we detected a strong IFN-γ response against K264 and HOK264 while at day 35 postimmunization the level of IFN-γ secretion following restimulation with K264 and HOK264 peptides was lower.

The system shows a period of low viral load after the initial phase of disease with is interspersed with small outbreaks when a new viral mutant occurs - until eventually the viral load rises again.

At the initial phase of disease, cell infiltration in the joints, cartilage proteoglycan loss, synovial TNF-α and soluble RANKL levels are similar in wild-type and properdin-deficient mice.

The results of the study will provide knowledge regarding aspects of COPD which are little known, such as the natural history of the initial phases of the disease and the impact and incidence of the exacerbations on the patients with mild-moderate forms of the disease attended at a primary healthcare level.