Sentence examples for inhibitory gating from inspiring English sources

Fig. 1 The inhibitory gating variable, s and the inhibitory current, I syn.

In contrast, enhanced occipital alpha has been related to an inhibitory gating mechanism regulating the intake of information in task-irrelevant visual areas [37] [42].

Such inhibitory gating processes include (i) blocking of ADP release by the "tethered" head until nucleoside triphosphate binds to the "bound" head (9, 11, 21) and (ii) slowing of ATP binding to the leading head when the neck linker in the trailing head is docked (22, 23).

The L97D mutant, on the other hand, mitigates Ca2+ deactivation and maintains a fairly high open probability (Po ∼ 0.4) even in the presence of 3 mM Ca2+, suggesting that this inner helix mutation favors the pore in an open conformation and thereby weakens the inhibitory gating effect of Ca2+.

Our present data demonstrate that coincident activity of converging cortical and subcortical drivers can overcome this inhibitory gate within a well-defined time window.

From a functional point of view it was often reported that layer IV acts as an inhibitory gate for incoming sensory information and that the basic mode of information processing in layer IV is inhibitory (Welker et al. 1993; Pinto et al. 2003).

Here, we show for the first time that in the HCN2 channel cGMP can also exert an inhibitory effect on gating via cGMP-dependent protein kinase II (cGKII -mediated phosphorylation.

Diminished inhibitory sensory gating also occurs in cocaine addicts, probably because of their increased catecholaminergic neurotransmission, which blocks the inhibition.

The exact neuronal mechanisms, however, have not been extensively investigated yet by delicate single unit recording methods Prepulse inhibition (PPI) of the startle response is a broadly used important tool to investigate the inhibitory processes of sensorimotor gating.

The difference between the plasticity produced by reducing the activity of PV- (Kuhlman et al., 2013) or SST- neurons during MD suggests that different inhibitory circuits are engaged in gating distinct aspects of cortical plasticity.

It also provides evidence that glycine inhibitory dysfunction gates tactile input to nociceptive specific neurons through PKCγ-dependent activation of a local, excitatory, NMDA receptor-dependent, circuit.