Altogether, our findings support a paternal inheritance model where the sperm provides two functional centrioles, of varied structure and composition, along with the associated PCM/striated columns structures.
Table 5 shows the relationship between CXCL9-11 polymorphisms and liver stiffness under a recessive inheritance model.
A genetic inheritance model may provide a more parsimonious account of these data than does a conditional adaptation model derived from sociobiology.
In our study, we found that CXCL9-11 polymorphisms, under an overdominant inheritance model, were associated with lower LSM values and fibrosis stage.
We selected the recessive inheritance model (CC vs. GG/CG) for the genetic association study, because it was the model that best fit to our data.
The overdominant inheritance model was the model that best fitted to our data, and the association values are shown in Table 4.
These analyses were performed in the additive inheritance model.
This was not unexpected since rs4961199 was initially identified using a recessive inheritance model [12].
A dominant inheritance model is assumed, and we neglect the probability of an individual exhibiting two rare variants.
We have additionally tested the inheritance model of these two polymorphisms by the Bayesian model-free approach [106], [107].
Due to the small sample size of the IVGTT subgroup, we performed these analyses in the dominant inheritance model.
