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Furthermore, although most local surveillance data are aggregated, the spread of influenza likely varies according to population subgroup.
The pathologic series of events leading to death from influenza likely has hereditary [ 18], environmental [ 19], and inflammatory [ 7, 9] components.
This survey was based on symptom recall rather than symptom reports, which may reflect the participants' enduring perceptions of influenza, likely to guide their behaviour in future influenza epidemics.
More recent epidemiological analysis support this finding (Shaman et al., 2010) and suggest that the marked seasonality and generally predictable geographical trajectory of seasonal influenza likely reflect, at least in part, climate variables.
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Among children under 2 y, S. pneumoniae and H. influenzae type b (Hib) are estimated to cause 36%and22%2% of radiological pneumonia cases respectively [ 18, 19], but other pathogens, including Staphylococcus aureus, Mycobacterium tuberculosis and non-typeable H. influenzae, likely play a substantial though poorly understood role [ 12, 13, 17].
The antiviral, like influenza, is likely still effective in the prophylaxis and treatment of disease caused by a new pandemic virus.
Thus, the clinical syndrome of influenza-like illness is likely to be less sensitive than that described here.
17 In pandemic influenza, however, a greater proportion of children presenting with influenza-like symptoms are likely to have influenza, which would tend to increase the apparent efficacy of neuraminidase inhibitors in clinical cases.
But a second wave of influenza would likely begin in four months, at the end of the summer.
"We don't know why that is, but the fact we have neither a vaccine nor experience being infected with this strain of influenza are likely explanations".
Ascertainment bias for the primary outcome of laboratory-confirmed influenza is likely to be minimal and non-differential.
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