"inflammatory factors" is a correct and usable phrase in written English.
It is a technical term used in the medical and scientific fields to refer to substances that cause inflammation. For example: "When the body's inflammatory factors become overactive, it can lead to chronic inflammation and pain."
The visceral fat — the fat located in our abdomen — churns out inflammatory factors and hormones.
They then attacked neurons, causing visible damage and significantly elevating levels of inflammatory factors.
The same inflammatory factors enhanced bone resorptive osteoclastic activity.
P2X7R expression in PDLSCs was demonstrated at both the gene and protein levels, whereby P2X7R expression was decreased in the presence of inflammatory factors but significantly increased in an osteo-inductive environment in the absence of inflammatory factors (Fig. 1).
The chronic protective effects of limb remote preconditioning and the underlying mechanisms involved in inflammatory factors in rat stroke.
Fig. 1: P2X7R expression in PDLSCs under normal/osteogenic conditions with or without the addition of inflammatory factors.
SASP is the secretion of pro-inflammatory factors into the microenvironment by a senescent cell.
HMGB1 does not increase the cytokines of pro-inflammatory factors, TNF-a and IL-1β, while RAGE blockade attenuated LPS-induced pro-inflammatory factors in primary spinal neuron culture.
However, HMGB1 does not increase cytokines of pro-inflammatory factors but promotes MAP-2 expression in primary spinal neuron.
mRNA levels of pro-inflammatory factors, including IL-6, IL-1β, CD68 and MCP-1 were not changed (Fig. 2B).
Likely, this is secondary to ameliorated disease states and balanced expression of growth factors and pro-inflammatory factors.
