RAP probably combats renal inflammation through dephosphorylation of lipopolysaccharide and adenosine triphosphate [122, 123].
Attenuating inflammation through direct removal of inflammatory mediators from circulation appears to be intuitively.
Chrysin significantly decreased inflammation through inhibiting NF-κBp65/IKK-β expression and TNF-α level.
When delivered by IV, SVF cells are able to recruit into areas of inflammation through the vascular system.
Targeting endothelial adhesion molecules may provide a more specific way to image inflammation through the detection of endothelial activation.
Local hypoxia can in turn promote inflammation through the release of DAMPs from host epithelial cells [35, 120, 121].
μ-Opioid receptor activation can modulate thermal hypersensitivity associated with tissue inflammation through the TRPV1 channels [69].
We hypothesized that watermelon would increase antioxidant capacity and reduce blood lipids and inflammation through modulation of related gene expression.
Macrophages cause inflammation through the production of interleukin-1, interleukin-6, and TNF-alpha.
In chronic fatigue syndrome and major depressive disorder, Leaky Gut Syndrome activates the intestinal immune system to produce chemicals called cytokines that spread inflammation through your body.
Endogenous AGEs cause inflammation through interaction with specific receptors (RAGE) [13].
