Sentence examples for inflammation stimulation from inspiring English sources

Neuronal injury in infection can originate from systemic inflammation, stimulation of local immunocompetent cells and direct action of bacterial toxins on neurons [ 1, 18].

During a sterile insult of inflammation stimulation, hypercapnia may be of benefit but would be deleterious in the setting of infection due to host immunosuppression.

It has multiple pharmacological effects including; antimicrobial activity against 54 microorganisms [ 19- 21], inhibition of intestinal ion secretion and smooth muscle contraction, inhibition of ventricular tachyarrhythmia, reduction of inflammation, stimulation of bile secretion and bilirubin discharge [ 22].

Low-level laser (light) therapy (LLLT) is a rapidly growing alternative approach to many medical conditions that require relief from pain and inflammation, stimulation of healing, and prevention of tissue death after injury or infarction [ 4].

Previous evidence indicates that exogenous APC promotes healing of recalcitrant ulcers in patients with diabetes, acting via numerous different mechanisms including inhibition of inflammation, stimulation of angiogenesis, and reepithelialisation [ 8, 14, 15].

So anyone wanting these products for their animal's chronic pain, anxiety, inflammation, appetite stimulation, or epilepsy have to live in a state where medical marijuana is legal – and even then, they need to have a prescription for themselves just to enter a dispensary.

Following 8 week oral treatment with CFZ, mice exhibited decreased inflammation upon stimulation with carrageenan at t = 4 h (~3-fold), 24 h (~14-fold) and 48 h (~2-fold) (*p < 0.05), compared to mice fed with a control drug-free diet (Supplementary Fig. S6).

Our results show that inflammation and stimulation had more of an effect on HBD2 production than these genetic factors.

Thus, division I-1 strains are the most potent inducers of inflammation and stimulation of sebocytes and keratinocytes, the latter by selective induction of human β-defensin-2 and interleukin-8 through Toll-like receptors 2 and 4 [6], [6].

Several hypotheses have attempted to explain the noted pathology, but most now include cell adhesion to the endothelium or direct infected RBC (iRBC) interactions with platelets as promoting pro-thrombotic immune responses, resulting in further vascular inflammation, immune stimulation and obstruction of cerebral capillaries [2].

The time course of POAF unmasks the importance of temporary surgery-induced factors as inflammation, sympathetic stimulation, and oxidative stress.