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Since H.I.V. zeros in on activated immune cells, inflammation increases infection risk.
Systemic inflammation increases the risk of acute ischemic stroke [84].
The presence of inflammation increases the gingival fluid volume.
The acute peritoneal inflammation increases postoperative C-reactive protein concentrations (CRP) and causes postoperative pain [2].
Inflammation increases local vasculitis and thus decreasing the oxygen availability to inflamed areas [6].
Ocular inflammation increases capillary protein permeability allowing greater leakage of proteins into the vitreous space.
Inflammation increases with age, and the number of people reaching age 90 years and older is growing, making the association between inflammation and dementia increasingly relevant.
To test the hypothesis that intrauterine inflammation increases prostaglandin production and may be a risk factor for persistent ductus arteriosus after therapy with indomethacin, a nonselective cyclooxygenase inhibitor.
The purpose of this study was to assess whether inflammation increases vulnerability to hypoxia, and influences the effect of 100% O2 and 21% O2 reoxygenation on brain.
Moreover, its rapid translocation to the endothelial cell surface at the sites of inflammation increases its potential as an imaging target.
We hypothesized that exogenous glucose administration during inflammation increases cardiovascular dysfunctions by activating glucotoxicity pathways and that this is prevented by endothelial PTP1B gene deletion.
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CEO of Professional Science Editing for Scientists @ prosciediting.com