Sentence examples for inflammation generated by from inspiring English sources

Therefore, it is reasonable that GLP-1 should, by reducing oxidative stress generation, improve endothelial dysfunction and inflammation generated by both hyperglycemia and hypoglycemia.

Therefore, it is reasonable that GLP-1 should, by reducing oxidative-stress generation, improve endothelial dysfunction and inflammation generated by hypoglycemia.

Therefore, it is reasonable that, in our study, GLP-1 should, reducing oxidative stress generation, improve endothelial dysfunction and inflammation generated by hyperglycemia following hypoglycemia.

The neurogenic inflammation is generated by the release of neuropeptides such as calcitonin gene-related peptide (CGRP) from trigeminal neurons terminals and leads to inflammation with the release of cytokines such as interleukin-6 (Il-6) and tumor necrosis factor-alpha (TNF-α) [2].

However, these treatments are not completely effective in reducing the neurological alterations, as they cannot eliminate hyperammonemia or inflammation continuously generated by the liver disease (14, 30, 35).

Although LXs are AA-derived eicosanoids, they can be clearly distinguished from the classical proinflammatory prostaglandins, thromboxane, and leukotrienes on the basis of their capacity to trigger a self-limiting response to inflammation when generated by leukocytes.

Additionally, leukocytes activated by septic inflammation generate reactive oxygen species that directly disrupt microcirculatory structures, cellular interactions, and coagulatory function [ 24- 26].

Further studies are required to determine whether curcumin can reduce inflammation and degeneration generated by combinations of different catabolic stimuli.

It appears that a state of constant inflammation can be generated by some patients in response to some of these nondegradable materials.

In the progress of inflammation, NO is generated by activated inducible iNOS, and participated in the innate response along with other macrophage mediators in many mammal.

Here we demonstrate that intracellular stress induced by glucose deprivation promotes tubular inflammation generating a proinflammatory microenvironment prone to the development of alloimmunity.