Sentence examples for inflammation augment from inspiring English sources

It is possible that altered secretory patterns associated with gastric inflammation augment the development of obesity-associated conditions.

Chronic stress has also been associated with hypertension [ 20] and may interact with lead to stimulate the hypothalamic pituitary adrenal (HPA) axis, promote oxidative stress and inflammation, augment adrenergic activity, and alter the renin-angiotensin system [ 18, 21, 22], which are critical to blood pressure regulation.

When challenged with either intratracheal zymosan or LPS, NADPH oxidase-deficient p47phox−/− mice and gp91phox-deficient mice developed exaggerated and progressive lung inflammation, augmented NF-κB activation, and elevated downstream pro-inflammatory cytokines (TNF-α, IL-17, and G-CSF) compared to wildtype mice.

The mixture of cytokines, mimicking the in vivo cytokine load in islet inflammation, augmented the responses seen with the individual cytokines.

During an exacerbation airway inflammation augments and a sustained worsening of patient's condition from the stable state and beyond normal day-to-day variations is observed.

Although we did not study the functional correlation of sepsis-induced CML depositions and myocardial inflammation augmented by MV, it seems logical to hypothesize that this could contribute to sepsis-induced myocardial dysfunction.

Since IL-1β actions on glomus cells of the carotid body mimicked the effect of hypoxia, we sought in vivo evidence that systemic inflammation augments carotid body chemosensitivity and increases ventilation.

In turn, an increase in the gastric production of proinflammatory cytokines may contribute to systemic inflammation and augment inflammatory processes in hepatic parenchyma.

These data suggest that therapeutic strategies that antagonize TNFR1 to limit inflammation and augment TNFR2 signaling to enhance remyelination might provide effective treatments that both ameliorate disease and promote recovery in MS patients.

PUFAs and lipoxins, resolvins and protectins suppress A-FABP expression, inhibit macrophage and cyclooxygenase-2 activation, and decrease production of proinflammatory cytokines, and thus decrease insulin resistance and resolve inflammation and augment recovery from sepsis [ 18, 19].

We propose that Th17 plasticity contributes to the persistence of joint inflammation by augmenting local levels of GM-CSF.