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Their research aims to show complex interactions of biological contagion and infectious sequelae among individuals who are interconnected in networks of various degrees of separation across the globe.
However, of most concern are the post infectious sequelae of rheumatic fever/rheumatic heart disease and chronic renal disease.
As a consequence, there is under- or over-estimation of Campylobacter-triggered post infectious sequelae.
These patients have presence of thrombocytopenia, eczema, immunodeficiency, and infectious sequelae.
Careful and frequent surveillance (eg, with quantitative polymerase chain reaction), prevention, and treatment of these infectious sequelae are critical in the care of HSCT recipients with WAS.
Although some degree of acneiform rash occurs in most patients, severe eruptions resulting in significant pain, pruritus, or infectious sequelae are rare.
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GAS is also associated with post-infectious sequelae, including rheumatic heart disease and glomerulonephritis.
Although this acute enteritis is self-limiting, post-infectious sequelae GBS, RA and IBD can arise after recovery [ 1– 3].
While mortality is low, morbidity imparted by post-infectious sequelae such as Guillain-Barré syndrome, Reiter syndrome/reactive arthritis and irritable bowel syndrome is significant.
In consequence of acute enteritis, extraintestinal post-infectious sequelae, namely, the Guillain-Barré syndrome, inflammatory bowel disease, and reactive arthritis may occur [ 3, 4].
Campylobacter jejuni and C. coli are the most common bacterial cause for gastroenteritis in industrialized countries [ 1] and have been implicated in the development of several post-infectious sequelae [ 2, 3].
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