Sentence examples for infections persistent from inspiring English sources

Patients with the deficient expression of selectin ligand by neutrophils suffer from recurrent infections, persistent leukocytosis and severe growth and mental retardation.

Although type I interferon (IFN-I) is thought to be beneficial against microbial infections, persistent viral infections are characterized by high interferon signatures suggesting that IFN-I signaling may promote disease pathogenesis.

For several decades, numerous circumstantial data, retrospective epidemiological or clinical reports demonstrated that chronic infections, persistent injuries or inflammation induce precancerous state of tissue that increases the risk of many cancers, particularly in aging individuals [37 39, 44, 45].

While the majority of cervical HPV infections are transient infections, persistent infections are significantly more likely to progress to both premalignant and malignant lesions[13].

Unlike acute infections, persistent infections are difficult to treat owing to their complex biology which involves host-pathogen interactions at various levels ranging from molecular to population levels and maintenance of such interactions for prolonged time adds further complexity [19].

Congenital disorder of glycosylation, type IIc (CDG IIc), also known as leukocyte adhesion deficiency II (LAD II) or Rambam-Hasharon syndrome (RHS), is an autosomal recessive syndrome, characterized by recurrent infections, persistent leukocytosis, severe mental retardation and slowed growth [1], [2].

Resistance and super infection: persistent responsible pathogen with developing of another pathogen.

In HIV-1 infection, persistent HIV-1 specific IFN-γ+ CD8+ T cell responses are detected in the setting of disease progression, consistent with functional impairment in vivo.

The methods used in this study do not permit us to conclusively distinguish between incident infection, persistent infection with intermittent viral reactivation, and passive shedding of viral nucleic acids associated with latently infected cells in the absence of viral replication.

Directly or indirectly, infectious agents produce long-term outcomes through pathways that include acute infection, persistent active infection, persistent nonreplicating (latent) infection, immune response to an infectious agent that may not commonly be pathogenic, and malignant transformation.

The probable reasons for this controversy might be due to several factors including re-infection, persistent infection, persistent exposure to mycobacteria, and possible maintenance of the circulating pool of effector memory T cells, rather than technical factors [ 7].