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The data reported here provide specific insight into the antigens that are recognized by the humoral immune response after natural infection(s) in a naturally infected cat population.
Diseases such as cat distemper are spread by viruses that can lie dormant in the environment for weeks, so a cat doesn't need to have direct contact with an infected cat to pick up infection or illness.
As previously reported for most infected dogs from the same area, the infected cat lacked apparent clinical signs of infection, suggesting that subclinically infected animals might be carriers and reservoirs of O. lupi nematodes (7 ).
Most FIV infections are acquired as a consequence of bite wounds inflicted by an infected cat, presumably through inoculation of virus or virus infected cells [ 16, 17].
One antigen (BH11090) was found to be significantly more reactive in the type II infected cat group than in the type I infected group (p-value = 1.7×10−2).
T. gondii parasite preparations were generated from enterocytes from an infected cat at onset of patency (5d post infection).
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Single FIVC infected and dual infected cats separate along the second axis.
This profile discriminates dual infected cats from cats infected with FIV alone, which show high IL-10 and lower numbers of CD8 and CD25 cells.
Similarly, humans could be similarly infected by contact with infected cats at the animal shelters.
It is likely that this was the source of infection for the two infected cats in this study.
Gardening in cyst-infested soil, handling infected meat, or emptying litter boxes used by infected cats can result in infection.
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