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A number of rather small studies produced controversial results when patients were treated in the setting of an acute myocardial infarction to limit infarct size and improve wound healing [ 88].
Accordingly, Hs-CRP can be as an independent risk factor for cerebral infarction to predict the severity of cerebral infarction and infarct size.
The remaining physicians varied in their answers from "no infarction" to "certain infarction".
GRO and Il-6 act together following infarction to improve cardiac function through the reconstitution of tissue mass.
New solutions are needed to regenerate hearts damaged by myocardial infarction, to overcome bad prognosis of patients with heart failure, and to address the shortage of heart donors.
Thus it has become one of the target for treating cerebral infarction to decrease blood viscosity by integrated Chinese and western medicine.
A gene manipulated cell patch using a homologous peritoneum substrate was developed and applied after myocardial infarction to repair scarred myocardium.
In these cases, there was a large difference in the interpretations made by the referring physicians, and they used the whole range from "no infarction" to "certain infarction" for their interpretations.
The biodegradable cardiac patches with or without AAV-GFP were implanted over rat left ventricular lesions three days following myocardial infarction to evaluate the transduction effect of released viral vectors.
In spite of the potential advantage of computational speed in [25], the effect of infarction might not be reflected by a small number of principal modal components because the influence of infarction to the whole myocardium could be localized and small.
This cardiac OT-GKR differentiation is important in the development of cell therapies for hearts injured by infarction, to induce the cardiac differentiation of somatic stem cells in diseased adult hearts for their regeneration.
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