The prevalence of failure to regain birth weight among LBW infants was calculated by dividing the number of infants who had not regained their birth weight at 21 days of age by the total number of infants enrolled in the study.
Finally, sources of infection for infants were calculated from published data [29], [32] and are presented in Table 2, along with other key parameters used in the epidemiological model.
Multiple pregnancies with two or more live infants were calculated as one.
Mortality rates, presented as deaths per 1000 liveborn infants, were calculated as the proportion of deaths among infants at risk.
Risk-adjusted centre variations of interventions and morbidities among high-risk infants were calculated using a multilevel analysis.
Z scores for length, weight, and head circumference measurements of infants were calculated with a computerized program that was developed for Turkish children [ 29, 30].
Based on the differences in incidences in infectious morbidity (76% and 50% respectively) in the GEEF study [ 22], and a two-tailed α = 0.05, β = 0.20, a sample size of 2 × [2*7.85*0.63 0,37)]/ 0,26 2 = 2 × 54 infants is calculated.
By employing the above prevalence of 12.8%, 40% of Thai infantile cholestatsis being have idiopathic cholestasis [ 24] (and our unpublished data), and panethnic incidence of cholestatic jaundice 1/2,500 infants (no specific data for Thai infants) [ 1, 2], the incidence of NICCD among Thai infants is calculated to be 1 in 48,828.
The equivalent weights of the PCs representing the tactile and nociceptive-specific potentials in full-term infants were calculated at Cz in 60 EEG epochs (30 in term and 30 in preterm infants) following time-locked touch or noxious lance of the heel of 41 infants, aged 28 45 weeks GA (born at 24 41 weeks GA).
