Sentence examples for inefficient suppression from inspiring English sources

Inefficient suppression may lead to weak audiovisual suppression in migraineurs compared with normal controls.

Thus, underexpession of full-length CbfA protein in JH.D cells is not due to cbfAam mRNA instability, but is rather a consequence of inefficient suppression of the premature stop codon.

Notably, inefficient suppression of platelet activity upon treatment with clopidogrel and ASA has been associated with significantly increased cardiovascular risk [ 9, 10].

As the anti-inflammatory cytokine IL-10 suppresses the macrophage-dependent synthesis of both TNFα and IL-1 in nonmalignant conditions, it was most interesting to see that the responses to IL-10 are dysregulated in RA macrophages, resulting in an inefficient suppression of inflammation [ 22].

Our findings are consistent with a model in which low expression of IL-28 family cytokines during allergic inflammation and/or viral infection predisposes asthmatic patients for exacerbation of allergic airway disease both via reduced viral clearance and via inefficient suppression of Th2 and/or Th17 responses.

Upon ectopic expression of Abd-B.r together with cad, which reflects their native co-expression in PS15, we observed almost the same efficiency in suppression of NB7-3 (91%, n=96 hemi-PSs; Fig. 8E, Fig. 9B) and of other NBs (25.43±1.50, n=14; supplementary material Fig. S6D; Fig. 8G), as opposed to the inefficient suppression of NB7-3 upon misexpression of Abd-B.r alone.

This step enhanced the specific signal by reducing (1) the C-terminally truncated CCR5 receptor population that could arise naturally or as a result of inefficient amber suppression and (2) the nonspecifically labeled proteins other than CCR5 that appeared in the Western blot as multiple nonspecific bands.

First, it opens the possibility that for some tumors, lack of response could be due to inefficient tissue estrogen suppression, not detected by total body tracer studies or plasma estrogen measurement.

Additional manganese improved the ability of cytosolic Sod2 to suppress the sod1Δ mutation (Fig. 3D) suggesting that the weak suppression is due to inefficient metallation of the Sod2 protein in the cytosol.

This neurodegenerative phenotype occurs subsequent to early-life suppression of DAT, suggesting that inefficient DA re-uptake might contribute to these behavioral and histological changes.

In contrast, it has been suggested that HIV-immune dysfunction is associated with suppression of HIV-specific effectors by Tregs, leading to an inefficient immune response against HIV [36], [39], [45], [46].