Sentence examples for induction to control from inspiring English sources

This combinatorial vector employed a hypoxia-responsive promoter and repeated targeting sequences of an miRNA that is enriched in NSCs but downregulated upon hypoxia induction to control the transgene expression.

In our IgG-depletion experiments on ACPA-positive and -negative RA cases, we observed a marked reduction of NET induction to control levels in both cases, whereas reconstitution of serum with IgG gained from depletion almost completely restored NET induction in the ACPA-positive cases.

However, high-risk patients (WBC >10 000/μl at presentation) achieved an inferior CR rate of 79 81% because of early treatment failure from fatal hemorrhage and differentiation syndrome despite the addition of either gemtuzumab ozogamicin (GO) or idarubicin during induction to control elevated WBC counts.

Accompanying the extension of the excretion time (after 12 or 24 h induction), compared to control cells, recombinant cells are more sensitive to erythromycin and deoxycholate, and have a slightly increased sensitivity to lysozyme (Fig. 1e).

In contrast to the in vivo measurements, spiked sediments showed significantly elevated EROD induction compared to control sediments in an in vitro assay using the permanent cell-line RTL-W1.

Finally, by using T7 induction system to control the expression of both small RNA and chaperone Hfq, a small RNA regulatory tool was developed and optimized to be a strictly inducible off system for gene regulation in Synechococcus 2973.

Expression of the gene of interest was divided by the house keeping gene glyceraldehyde-3-phosphate dehydrogenase (GAPDH) and expressed as fold induction compared to control.

In histological evaluation 72 hrs after ALI induction, compared to control animals, saline-treated mice demonstrated 8.4 ± 1.3% necrotic area, mild regeneration and extensive infiltration of inflammatory cells (260 ± 40 per high power field, n = 6) in the centrilobular region.

In histological evaluation 48 hrs after ALI induction, compared to control animals, sham IgG treated mice demonstrated 14.6 ± 1.2% necrotic area and extensive infiltration of inflammatory cells (270 ± 40 per high power field, n = 9) in the centrilobular regions.

Insufficient glucose supply after induction was observed to control acetate accumulation effectively and improved the expression of the target gene evidently.

The induction was reduced to control levels after 4 h of treatment.