Sentence examples for induction findings from inspiring English sources

The incidence of acute rejection was significantly reduced in the presence of rATG (23%% vs. 55%%; p = 0.03), with a non-significant reduction in the incidence of BOS (20 % with rATG vs. 38%% vs. no induction), findings that were maintained long-term [ 170].

RCTs by Garry et al [ 80] [LOE: 1-] and Lokugamage et al. [ 81] [LOE: 1-] compared vaginal misoprostol versus vaginal prostaglandin E2 inserts for cervical ripening and labour induction, finding no significant differences in perinatal outcomes.

We also note that despite the absence of SIRT1 induction, our findings are consistent with a general alteration in the expression of genes involved in chromatin remodeling by CR and resveratrol, including other sirtuins (Figure 5).

Since beclin-1 needs to be exported from the nucleus to the cytoplasm in order to play its function in autophagy induction our findings further support the hypothesis about the differences between the outer population of cells receiving direct signals from the substratum and the centrally located cells lacking the contact with ECM, thus inducing autophagy and being prone to apoptotic cell death.

Deterrence of natural populations of herbivores has only rarely been observed in previous studies, because most have used captive trials and tested for reduced herbivore performance as the measure of induction, sometimes finding no effect on specialists [16].

Our study found that overweight and obese women were more likely to have induction, a finding reported by other studies [ 4, 9, 10, 14, 20– 22, 30, 58].

Consistent with these findings, induction of CD14 transcription in brain tissue of susceptible mice exhibiting symptoms of ECM has recently been described by our group.

Because the authors also found that METH, itself, also caused NQO1 induction [104], their findings and our present observations indicate that toxic doses of the psychostimulant activate a protective cascade which is mediated via Nrf2-regulated transcriptional events, which involve coordinated induction of several genes that code for phase II detoxification enzymes including Hmox1 and NQO1 [101].

Based on these findings, induction of PUN1 upon ion stress can be partly explained.

In concordance with these findings, induction of a stress response, which acts mostly via the same signal transduction molecules as TNF, inhibits apoptosis [ 60].

Consistent with the present findings, induction of chemokines has been previously described after stimulation of chondrocytes with IL-1 [ 56, 57], but their suppression by EGCG has not been previously described in OA chondrocytes.