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Two-way ANOVA was used to analyze data from open field and light-dark box tests as Q-Q plots indicated these data were normally distributed, and the variances between treatment groups were homogenous.
Except where indicated, these data were obtained from the Broad Institute, MA [ 49] and represent approximately 2-fold statistical coverage of the genomes.
To determine the missing data mechanism, we conducted a missing completely at random (MCAR) test (Little, 1988), which indicated these data were missing at random, χ(2719) = 2583.17, p > 0.05.
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This power analysis indicates these data sets are large enough to detect relatively small differences in somatic aging spans.
Loss of antigenicity as indicated by these data suggests immune evasion by the parasites.
Participants considered clinical information most often when assessing violence risk, indicating that these data were most readily available.
Larger randomized controlled trials are indicated to confirm these data.
These data indicated that ROS production was not disease-specific and a function of age.
These data indicated that NS1 suppressed Rac1 activity by inhibiting K63-linked ubiquitination.
These data indicated that loss of methylation in late-replicating domains reflects mitotic activity.
These data indicated that the high-quality transcriptomic reads were suitable for downstream analyses.
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CEO of Professional Science Editing for Scientists @ prosciediting.com