In our preliminary studies comparing PRL-induced tumors to other murine models of breast cancer, PRL-induced ERα- and ERα+ tumors clustered together, indicating shared features suggesting a common origin.
As shown in Figure 3, there are 28 common enriched pathways (CEPs) across all four subtypes of breast cancer (See Additional file 5 for the whole list), which indicate the shared features of breast cancer.
Antelman interpreted these ubiquitous cross-sensitization findings to indicate that the shared feature of the drugs, food, or environmental stimuli was their quality as novel and threatening stressors, i.e., individually salient "danger signals" for the organism, rather than their pharmacologically specific actions [ 187].
This result indicates that perirhinal cortex atrophy disrupts the disambiguation of inherently confusable living things, which consist of a large number of shared features and few distinctive features, compared with non-living things, which consist of fewer features overall and a greater proportion of distinctive to shared features (Tyler and Moss, 2001; Cree and McRae, 2003; Randall et al., 2004).
They found other shared features as well.
Strikingly these two genes have shared features.
Our studies indicate that nucleotide binding is a shared feature of multiple MF transporters, though this feature appears to have arisen separately in each of the proteins.
It appears that a high proportion of Cajal body members are positively identified by the nucleolus predictor (12/36 = 33% of all Cajal body proteins) indicating that nucleolar proteins may share features with the small set of Cajal body proteins.
These results indicate that these two proteins may share features of their function and regulation.
Although it shares features with other recently described lobopods, it differs in possessing lateral lobes along the body with dorsal gill-like structures attached to them, a feature that may indicate a relationship with the enigmatic Opabinia4.
The overlap of the clinical features and genes indicate that shared common pathogenic mechanisms may contribute to the liability for both ASD and schizophrenia.
