Plastic strain amplitude and hysteresis loop area are found to decrease with increasing mean stress.
For the baseline simulations we present, we assume that the DR strain has a fixed moderate fitness cost which reduces both the probability of transmission and the probability of progression after infection by 20% each; in further analyses, we discuss how an increasing mean relative fitness among DR strains over the course of a drug resistant TB epidemic would affect our results.
The small family of FPRs have increased mean abundances in lab strains, but the increase is not significant and largely the consequence of one unusually variable outlier, Fpr-rs6 (Two-way ANOVA, variance in receptor class F2,28 = 0.312, P = 0.734).
Lifespan effects also varied, with the gk131 strain showing increased mean lifespan and time to 90% mortality, whereas ppt-1 RNAi showed a decreased median lifespan.
Modulation of glyoxalase I activity was found to affect lifespan of transgenic worms (i. e., overexpression of CeGly1 leads to an increased mean lifespan of +29% whereas strains with a down-regulated CeGly1 are short-lived [-52[-52%
A noteworthy nuance is that some deletion strains (∆yebA, ∆ynfM, ∆yeeA, and ∆yebS) markedly increased homogeneity of the population with IMP-GFP signal, but only ∆yebA simultaneously increased mean IMP-GFP signal intensity (Fig. S5a).
Illustrative numerical results are presented and it is shown that an approximately sixfold increase in a mean strain rate function is required to displace the flame from a location where flashback to the nozzle is about to occur to a position so close to the wall that extinction is imminent.
The changes of strains recorded during the tests show higher maximum mean strain for the case, where the mean load increases in block.
Eventually, strain distributions were obtained in terms of mean strain and strain amplitude.
