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Increasing data suggest that histologic type substantially impacts STS behavior.
Increasing data suggest that errors in medicine occur frequently and result in substantial harm to the patient.
Increasing data suggest that the adoptive transfer of CMV-specific CTL constitutes an effective strategy against CMV infections.
Increasing data suggest that ischemic heart disease (IHD) shares several characteristics with common inflammatory diseases (such as rheumatoid arthritis), in which the pathogenetic role of inflammatory gene polymorphisms is well established.
Increasing data suggest that tumor dormancy can be regarded as a protracted asymptomatic stage during which tumor cells either remain in a quiescent state or their proliferation is balanced by cell death due to immunosurveillance or insufficient angiogenesis (Aguirre-Ghiso, 2007; Giancotti, 2013; Hensel et al., 2013) (Fig. 1).
Nevertheless, increasing data suggest that there are non-phosphorylated receptor substrate interactions that may affect ErbB-mediated responses.
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Increasing data suggests that they have an increased risk of progressive chronic kidney disease, cardiovascular morbidity and premature death.
Increasing data suggests that collagen-producing myofibroblasts that accumulate in the peritoneum in the course of PD are derived from various precursors.
However, recent evidence suggests that complement activity transcends innate host defense and there is increasing data suggesting complement as a regulator in processes such as allo-immunity, stem cell differentiation, tissue repair, and progression to fibrosis.
For minor oral surgery such as teeth extraction, increasing data suggests that continuing antiplatelet medication during oral surgery does not increase the bleeding risk notably whereas the role of local hemostasis is emphasized [ 13, 14].
In addition, there is increasing data to suggest direct tumor promoting effects of DKK-1.
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