Sensitivity, specificity and positive predictive value of increased central and peripheral vascularity in the diagnosis of LMS were 100, 86 and 19 %, respectively.
Zhang et al., found that Chinese people with type 2 diabetes had increased central and peripheral PWV compared to controls, and fasting glucose was a determinant of peripheral artery stiffness.
Moreover, a persistent noxious, mechanical, or thermal stimulation input can be involved in developing or increasing central sensitization and release processes, leading to or supporting chronic pain.
This symptom might reflect other common postmenopausal conditions such as irritable bowel syndrome, peritoneal adhesions, and increasing central obesity.
In our study, age was associated with increasing central obesity, systolic blood pressure, and prevalence of diabetes, all factors associated with the metabolic syndrome and NAFLD.
Histological changes in diabetic muscle included localized accumulations of PrP, Aβ, NOS1 and 2, and SOD1, and evidence of increased central nuclei and cell infiltration.
At pretask, a significant association between increased KSS and increase central beta and occipital alpha was found for the lesioned hemisphere.
These differences are not accounted for by differences in demographic characteristics (age, sex, and race/ethnicity), diabetes duration, and A1C but are largely mediated through increased central adiposity and higher blood pressure levels in youth with type 2 diabetes.
In contrast, controls showed a significant association between higher KSS and increased global theta power as well as increased central alpha and higher KSS at pretask.
In people with chronic OA, there is increased central excitability and decreased central inhibition [ 5].
