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The results indicated that the cell volume increased with the increases of activation time, whereas membrane surface adhesion force decreased, even though the local stiffness for resting and activated cells is similar.
Both TBI survivors and healthy controls had pronounced within-group increases of activation in areas of the DMN as an effect of TOT.
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Increase of activation energy for nonradiative deactivation of excited-centres luminescence decay can be achieved by changing the host crystal or by electron traps.
The breakthrough time first increased and then decreased with the increase of activation temperature.
The breakthrough time first increased and then decreased with the increase of activation time.
It was observed a decrease of activation energy with the increase of salt content till 10% and at higher salt concentration an increase of activation energy.
It is obvious that for NOAC, the adsorption capacity decreases slowly with the increase of activation temperature, reflecting the decrease of the pore volume.
A general task demand-independent increase of activation in AD patients compared with controls was found in right middle temporal gyrus.
Remarkably, fibres did not elicit any substantial increase of activation markers and inflammatory cytokines in dendritic cells, which remained in their immature (inactive) state.
With the increase of activation time and H3PO4 concentration, the adsorption capacity increased till mid concentration value of H3PO4, after that adsorption capacity decreased due to excess of chemical agent.
It is seen that with the growth of concentration of physically and chemically bound water, the increase of activation energy (E) is observed, and at concentration of 12.9%, it reaches its saturation.
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