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As the chain-branching activation energy increases, cells take a shorter time to form, and the ratio cell length over width decreases.
PTH increases cells of the hematopoietic lineage, including hematopoietic progenitor cells [5].
As φ increases, cells move to the baseline.
Thus, loss of TSC2 increases cells size and overexpression of the protein decreases cells size.
Paradoxically, however, as the duration of ischaemia increases, cells become adversely sensitive to restored blood supply, giving rise to additional damage on reperfusion (reperfusion injury).
However, there was other evidence to show that downregulated miR-125b increases cells apoptosis in ETV6/RUNX1 leukaemia REH cells without affecting the p53 expression level (Gefen et al, 2010).
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Also, we found that ORP5/8 overexpression increases cell proliferation.
ZnO induces oxidative stress, decreases viability, and increases cell death in Caco-2 cells.
Increased TOR signaling increases cell size at division [ 27].
In locked cells, stimulation increases cell functions, instead of proliferation.
Pharmacological augmentation of autophagy increases cell viability in vivo.
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