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Fig. S4 RIF increased life span of activated PDK-1 strain, pdk-1 mg142 pdk-1 mg142

Rapamycin also increased life span of male flies, less so than that of females.

The increased life span of neutrophils may be associated with increased tissue injury in these syndromes [ 12, 14- 16, 20, 22, 29].

The increased life span of transgenic flies expressing superoxide dismutase indicates that free radical-scavenging enzymes are sufficient to delay aging in Drosophila (Tower 2000).

The absence of the GluR2 subunit accelerated motor neurons degeneration in mutant SOD1 mice, whereas the induction of GluR2 expression increased life span of these mice [160,161].

This means that the increased life span of these strains may not be taken as evidence in support of the oxidative damage theory of aging.

Also, it deserves mentioning that metformin failed to increase life span of male F344 rats [ 27].

Mifepristone did not increase life span of virgin females or males.

Rapamycin could increase life span of weak insulin/Igf signaling (IIS) pathway mutants and of flies with life span maximized by dietary restriction, indicating additional mechanisms.

The cell number, their function and life span become largely modified [ 9], with increasing life span of neutrophils, reduced function of monocytes and reduced life span and function of lymphocytes.

Feeding of mifepristone for the first 20 days of adulthood was sufficient to increase life span of mated females, whereas feeding of mifepristone from day 20 onwards had no detectable effect, suggesting a critical period in young adulthood.