The occurrence of A. mellifera was positively associated with increases in flowering and native plants.
Since the 28 h DEX treatment gave increases in flowering gene expression, particularly FT, for all TG lines the DEX treatment was decreased to gain some insight into the kinetics of this induction.
Across all years (2006 13) there is an increasing trend in flowering probability in the removal treatment (trend contrast P = 0.00008) but no increase in the ambient treatment (Ptrend = 0.57).
While in most F2 populations r values tended to increase with increased variance in flowering time, P12 (Est-1 × Br-0), P15 (Br-0 × C24), P66 (Fei-0 × Col-0), and P129 (C24 × RRS10) formed a distinct group (left-side circle, Figure 1B) with small variances, but differing r values.
However, crude protein concentration increased (P =.02) as soaking length increased in vegetative orchardgrass, and decreased as soaking length increased (P <.001) in flowering alfalfa hay.
Maximum flowering as well as increase in flower bud diameter and subsequently increase in flower diameter indicates suitability of that growing medium, where organic matter in growing substrate and optimum amount of NPK manipulate plant growth and flower size (Kiran et al. 2007).
Other mixtures including SCT (T2), SST (T4), and SFT (T1) were statistically not different from each other with 23 to 30%% increase in flowers per plant compared to control (GS) (T0).
Polyploid plants also exhibited a typical increase in flower size characteristic of most polyploids of Arabidopsis (Dilkes and Comai, unpublished observations).
Third, in M. guttatus and F1 hybrids, ploidy appears to cause an increase in flower size, although the difference is not always significant, whereas autotetraploid M. nasutus have smaller flowers than diploid M. nasutus.
Flowering peaked in the dry season (March to May) and early wet season (June – September), with about a two- to four-fold increase in flower abundance over the late wet season (October-December).
BA treatment may also delay the formation of floral organs by inhibiting the transcription of the A-, B- and E-class of floral organ-identity genes, which would allow more time to generate more floral primordia in inflorescence meristems, along with activating the expression of CUC1, which would result in a significant increase in flower number in J. curcas.
