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Vγ1.1+Vδ6.3+ T cells share multiple characteristics with natural killer (NK) T cells including expression of the activation markers CD44, and NK1.1, and low expression of the immature T lymphocyte marker CD24.
A subset of Vγ1.1+Vδ6.3+, referred to as γδ NKT, share phenotypic and functional characteristics with NKT cells including expression of the transcription factor PLZF and a requirement for SAP-dependent [24], [37], [39], [48].
More recently, it has been shown that β-arrestins can translocate to the nucleus and regulate transcriptional events [ 33], including expression of the antiapoptotic protein BCL2 [ 34].
These rat iNS cells have all the key characteristics of primary NS cells including expression of the NS cell markers and the potential to differentiate into astrocytes, oligodendrocytes, and mature neurons with functional membrane properties.
In breast cancer, the EMT state has been associated with cancer stem cell properties including expression of the stem cell-associated CD44+/CD24-/low antigenic profile, self-renewal capabilities and resistance to conventional therapies.
However, recently another contributing factor has also been identified, because it appears that mitochondrial iron overload causes oxidative stress which leads to activation of genes that combat oxidative stress, including expression of the iron exporter ferroportin (32).
Similar(44)
Events influencing the early phase of innate immune response in gouty inflammation include expression of the triggering receptor expressed on myeloid cells-1 (TREM-1).
Attempts to prevent inclusion body formation and directly increase the solubility of recombinant proteins include expression of the protein of interest fused to a heterologous protein domain as a solubilizing agent [2].
The luminal B subtype has a cell proliferation signature that includes expression of the MKi67 gene.
Late events include expression of the HIV-1 genes, assembly of the virion, its release from the infected cell, and maturation into fully infectious particles.
Microarray analysis shows that, similar to the role of T-bet in Treg homing, the co-expression of T-bet and RORγt creates a hybrid gene expression program, which includes expression of the Th1 homing marker CXCR3 [ 44, 47].
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