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Treatment of patients with symptomatic hypermagnesaemia includes discontinuation of magnesium administration, use of supportive therapy and administration of calcium gluconate [ 6, 107].
Management of OH includes discontinuation of antihypertensive medication, adoption of physical counter- manoeuvres, and use of mineralocorticoid (e.g. fludrocortisone) and/or adrenergic agonist (e.g. midodrine) medications [ 24].
Standard management of drug induced skin rashes usually includes discontinuation of the suspected drug and avoidance of further exposure to this drug in the future.
The treatment of NMS includes discontinuation of contributing drugs and supportive therapy, but specific treatment with dantrolene, bromocriptine, nondepolarizing neuromuscular paralysis and benzodiazepines, among other such agents, has been reported only anecdotally.
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Depending on the particular injury, treatment may include discontinuation of the precipitating activity only, discontinuation of all training activities, or, for more serious injuries, crutches or surgery.
Before surgery is undertaken, initial patient management should include discontinuation of oral intake, institution of aggressive pulmonary toilet, appropriate antibiotics as indicated, and continued nutritional support either through an enteral or parenteral route.
If increased abdominal pressure is noted, it is important to take early measures, including discontinuation of arthroscopy, for preventing the progression of organ dysfunction.
Treatment alterations included discontinuation of OnabotA therapy (n = 2), addition of antiepileptic (n = 3) or SSRI (n = 9) medications and discontinuation of ongoing antiepileptic (n = 3) or SSRI (n = 1) medications.
The evaluation of this parameter is simplified by including discontinuation of treatment for any reason.
Other precipitating factors include discontinuation of or inadequate insulin therapy, pancreatitis, myocardial infarction, cerebrovascular accident, and drugs (10, 13, 14).
This improvement probably originated in multiple factors, including discontinuation of sedative agents and gradual restoration of the functional electrophysiologic activity of the diaphragm.
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