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The chemotherapy regimen includes agents such as doxorubicin, cyclophosphamide, and etoposide that can render the patient with side effects such as cardiomyopathy, haemorrhagic cystitis, and myelosuppression as well as secondary malignancy such as breast or thyroid cancer (Rubino et al, 2003).
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Potential treatment options could include agents such as ibudilast, minocycline and -naltrexone.
Laboratory evidence also supports the clinical evaluation of docetaxel-based combinations that include agents such as trastuzumab and/or estramustine.
These include agents such as contrast medium, opioids, temperature (hot or cold), and vibration.
Antiestrogen drugs include agents such as tamoxifen, toremefene, raloxifene and fulwestrant.
The latter would include latrunculin A and cytochalasin A but might also include agents, such as BHQ and thapsigargin that liberate calcium into the cytosol.
106 Other molecules, which are capable of activating latent provirus include agents such as the non-tumor-inducing phorbol ester prostratin.
These included agents such as proprionic acids (e.g., ibuprofen, naproxen), indole (e.g., sulindac) and enolic acids (e.g., piroxicam).
Steroidal antiandrogens were first developed in the late 1960s, can be distinguished by their physiologic progestational effects, and include agents such as cyproter-one acetate, megestrol acetate and medroxyprogesterone.
Many of these inhibitory molecules were originally developed for anti-cancer indications and include agents such as MEK inhibitors [ 8], obatoclax and gemcitabine [ 68], flavopiridol [ 69], anti-cytoskeletal drugs [ 70] and etoposide [ 71], among others.
They include agents, such as phorbol esters, formyl peptides-complement fragments, elastin fragments, fatty acids (leukotrienes) as well as many uncharacterized excretions of inflammatory cells themselves, which react with specific receptors on the inflammatory cells, and secreted proteins such as fibronectin.
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