Sentence examples for in which propensity from inspiring English sources

We used two approaches, namely, the creation of matched pairs based on propensity score and logistic regression analysis of outcomes in which propensity score participated as a variable.

Analyses were performed in which propensity scores were calculated indicating the likelihood of HT use based on a ordinal logistic regression model with HT as the outcome adjusted for the covariates in the multivariate model [ 34].

Of note, two of the propensity scores used (for both outcomes) included an important pair in which one propensity score was nested within a slightly larger propensity score identical to the original propensity score except for the addition of a single covariate (glaucoma diagnosis).

There are four different methods in which the propensity score can be used to minimize the effects of measured confounding: covariate adjustment using the propensity score, stratification on the propensity score, matching on the propensity score, and inverse probability of treatment weighting (IPTW) using the propensity score [ 1– 5].

Google is really not much different from age-old direct marketing, in which a propensity for product interest is discerned, and a pitch made.

In the second stage, the effect of surgical versus non-surgical treatment on outcomes was examined by GLM model in which the propensity score was used as continuous variable and was adjusted as a covariate along with all other covariates cA two-staged regression approach was used.

In the second stage, the effect of surgical versus non-surgical treatment on outcomes was examined by GLM model in which the propensity score was stratified into five bins (based on distribution of scores) and was adjusted as a covariate along with all other covariates.

While we have been motivated mainly by the human case, in which the propensity of some individuals to take unfair, long-shot gambles is most striking, a similar effect might occur in any species with overlapping generations.

This behavior could be quantitatively explained by a minimal stochastic model in which the propensity of origins to fire depends on both the gradual accumulation of a limiting, recyclable replication-fork factor and a fork-promoted initiation of new origins [26].

This is particularly pronounced in type 2 diabetes in which the propensity to fall is increased but bone mass is not necessarily low.

The striking heritability of RA, together with the presence of autoantibodies that precede clinical onset in a subset of patients, supports a pathogenetic model in which autoimmune propensity is long-established by the time joint inflammation is 'triggered' (Fig. 2).