Sentence examples for in vitro progression from inspiring English sources

We have previously reported that HKc/HPV16 progressively become resistant to the antiproliferative effects of TGF-β1 during in vitro progression through HKc/GFI and HKc/DR stages [ 21, 24].

This in vitro progression is possibly controlled by unidentified non-coding RNAs [ 58].

Overall, we found no loss of Smad2, Smad3, Smad4, and no increase in Smad7 during in vitro progression of HKc/HPV16.

We have identified a multigene methylation pattern acquired during this in vitro progression to malignancy that is detected in vivo in both premalignant and malignant lesions.

HKc/HPV16 are initially as sensitive to the cytostatic effects of TGF-β1 as normal HKc but become increasingly resistant to the antiproliferative effects of TGF-β1 during in vitro progression [ 21].

Previous studies in our laboratory demonstrated that HKc/HPV16 are initially as sensitive as normal HKc to the growth inhibitory effects of TGF-β1, but become increasingly resistant during in vitro progression [ 21].

This problem can be partially overcome by studying invasive processes in relatively inexpensive in-vitro tumor progression assays in which conditions can be accurately controlled.

Partially purified extracts from the leaves of transgenic tobacco plants inhibited in vitro growth progression of Plasmodium falciparum-infected red blood cells.

In vitro unfolding/refolding progression of human γD-crystallin has been extensively studied, and these studies indicate that the protein contains a partially folded intermediate population.

Furthermore, the chromosomal imbalances and a tumoral cell fusion with the murine stroma cells in the xenotransplant give clues to the spontaneous in vitro cell progression to the most aggressive astrocytoma phenotype.

It is important to note, however, that in vitro fork progression is 4 5 ntds/s, 5 10 times slower than in vivo measurements, and thus one or more of these proteins may be required to increase the rate of fork movement.