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Alternatively, reporter genes can be introduced into the germline or into fertilized embryos in those model organisms amenable to such manipulation (nematodes, Drosophila, zebrafish, Xenopus, or mice).
In those model systems, there is a much more prominent role for CD4+ T-cells, as opposed to PMNs or soluble CD28 as observed in the early phases of sepsis.
Identification of naturally-occurring TNR diseases in other organisms also expands the scope of possible study in those model organisms.
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In those models "potential output" is what the economy would produce if prices were perfectly flexible.
They can really do faith big-time in those models.
The material models used in those models were first validated with experimental results from published literature.
To reduce the emission of the transportation, the environmental costs are addressed in those models.
However, identification is likely to be a problem in those models.
The behavior in those models is expressed in terms of operations with pre- and postconditions.
But the lack of mucosal layer in those models makes the intestinal absorption prediction less accurate.
Explanatory variables used in those models are derived from ambient land use characteristics, road segment properties, and traffic flow characteristics.
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