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We examined its expression in the developing LRPs.
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OsPIN1c (formerly named OsPIN4) was reported to express in the developing LRP (Wang et al. [2009]).
Since the tuberculosis (TB) cases among HIV infected population result from the reactivation of latent bacilli, it would be useful to develop LRP that can detect dormant bacteria.
As the LRP developed, the GUS staining was clearly detected in the root cap of LRP (Figure 3D3-D5).
Here, it was demonstrated that NO promotes LR emergence of the LRP that were initiated during the treatment in the growing root (distal portion), but not in the already developed root (proximal portion).
Supernumerary incisor and molar teeth as well as fused and large molar teeth develop in Lrp 4 and Wise deficient mice.
The MicF sRNA is also known to bind to other targets including the global transcription factor Lrp, where it indirectly activates genes in the Lrp regulon via antisense base-pairing with the translation initiation region of lrp mRNA [ 44].
In the repressed state, Lrp binds the proximal site interfering with transcription and Dam methylates the distal site blocking Lrp binding.
Along the complete root, lateral organs can be of three different types: developing LRP, blocked LRP and emerged lateral roots.
When the LRP developed to a hemispherical shape, a weak GUS activity was detected in the center of LRP, indicating a weak expression of OsPIN1b at this stage (Figure 3D2).
In the nogo/position condition the three speaker groups generated comparable LRPs in that the nogo-LRP started and ended roughly at the same time, as indicated by similar nogo-LRP onsets and nogo decision latencies, respectively.
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