Sentence examples for in sqn from inspiring English sources

Our current model for FHE2 function is based upon a conserved increase in SQN mRNA levels in Ler-0 and Fe-1 compared to Col-0.

The presence of this insertion varies between Arabidopsis accessions [ 41], and may contribute to natural variation in SQN expression as detected for other genes tightly linked to transposon sequences [ 42].

Thus, BMIQ is assumption-free, as it does not require a separate normalization to be performed on selected subsets of probes that are matched for biological characteristics (e.g. CpG density), as done in SQN and SWAN.

Although changes in SQN expression alone cannot account for the drastic differences between zll phenotypes in Col-0 and Ler-0, it is likely that SQN forms part of an important pathway that contributes to ZLL function and FHE activity during stem cell development.

For instance, in SQN the normalization is performed on probe subsets defined by specific CpG characteristics (e.g. shelves, shores, CpG islands); however, multiple different definitions for say CpG islands exist (Gardiner-Garden and Frommer, 1987; Takai and Jones, 2002; Wu et al., 2010; Zhao et al., 2009).

In SQN-synchronization, the primary attribute, sequence number (SQN), is used to protect the HLR/HSS and the MS from replays and to limit the number of the full authentication sessions by mutual checking the value of SQN attribute separately stored in the HLR/HSS and in the MS. On the other hand, the dominant attribute in the counter-synchronization is the counter attribute.

Only synonymous SNPs are present in the SQN coding sequence between Ler-0 and Col-0, indicating that differences in enzyme amino acid sequence cannot explain differences in function.

In a reciprocal experiment, to address whether lower levels of SQN in ZLL-independent accessions such as Col-0 may be a reason for the absence of stem cell defects, SQN was ectopically expressed from the strong embryonic pAtRPS5a promoter in the Col zll ago10-1 mutant.

Mk. III went into extensive testing at No. 25 Sqn in May 1940 and another troubling problem was found.

In combination with a zll mutation, which allows AGO1 greater access to miR165/166, increased levels of SQN in Ler-0 enhance repression of AGO1 targets, such as the Class III HD-ZIPs, and lead to a high frequency of terminal stem cell differentiation.

Fraction (R + P G administered in I was more effective in causing SqM and SqN than the same fraction administered in BS/G.