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This work presents a method for ischemia segmentation in rat heart photos.
The present study was designed to investigate the potency of protosappanin A, one major ingredient of C. sappan L., in rat heart transplantation.
It is unclear if short-term and long-term intermittent hypobaric hypoxic challenges exert different effects on cytochrome c oxidase and Bcl-2 family in rat heart.
Of note, this treatment achieved more extensive improvement of cardiac function, with greater initial retention and survival of donor MSCs, compared to intramyocardial MSC injection in rat heart failure models.
At sub-lethal dose of mercury chloride (1.30 mg/kg body weight 45 days daily) administered in rat, heart tissue shows an elevated level of lipid peroxidation (LPO) content and simultaneously decreased level of cardiac marker enzymes.
However, it has also been detected in the mitochondria in rat heart and liver [34] [36].
Similar alteration of mitochondrial content has already been reported in rat heart after CH [6].
Similar(4)
Prevention of formation of C5b-9 has been found to reduce tissue damage in rat hearts perfused with human serum.
Suliman et al. for the first time demonstrated that lipopolysaccharide stimulates mitochondrial biogenesis in rat hearts in response to oxidative cell damage [126, 127].
Myocardial contrast echocardiography (MCE) can induce bioeffects in rat hearts by local activation of the contrast agent gas bodies.
Pro-angiogenic VEGF treatment increases the vascularization of the infarct zone in rat hearts following myocardial infarction (Mathison et al., 2012).
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