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These large corporations failed to qualify for the A-List because of weak results in our screen.
Three dct genes were found in our screen.
SFRP2 mRNA was also found increased in our screen.
Polo kinase, a SUMO substrate found in our screen, partially colocalizes with SUMO at both sites.
The genetic interactions identified in our screen appear to be especially important for regulating dynamic epithelia.
Interestingly, an srtD mutant (strain 29E04) was also attenuated in our screen.
Therefore, proteins whose depletion causes such alterations may have been missed in our screen (Figure 8A).
Using the fusogenic reagents found in our screen allowed us to characterise LD fusion.
This could explain why no BUD23 mutant was isolated in our screen.
However, none of the genes in the agf operon were identified in our screen.
Several hits identified in our screen are compatible with these findings.
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Justyna Jupowicz-Kozak
CEO of Professional Science Editing for Scientists @ prosciediting.com