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In our past study an innovative integrated orbit/formation model (IFC) has been designed, introducing a novel set of Hill-type equations.
In our past study we graphed RETT over inspiratory V' for ETTs with inner diameters of 6.5 to 9.0 mm [ 1].
In this study, we examined STZ-treated mice at 19 dpi, whereas in our past study in NOD mice, we examined mice at 40 dpi [ 17].
The model compounds and active agents used in our past study were of low molecular weight and relatively hydrophobic, and could thus be loaded into superhydrophobic coatings using a variety of organic solvents.
In our past study investigating the clinical features of 62 patients with positive ACA, only one patient had typical CREST syndrome, 9 patients had Raynaud's phenomenon (8 with sclerodactyly, telangiectasia or calcinosis), while rheumatoid arthritis (RA) was the most common diagnosis (21 patients) [ 2].
We determined that SLV-I would be less costly than practice-located vaccination if the estimated cost-per-vaccinated child for SLV-I were below the median/mean ($22.17/$39.54 in 2010 US $) cost-per-vaccinated child for practice-located influenza vaccination as reported in our past study [ 28, 30].
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We also recorded eye gaze position to monitor subjects' attention and galvanic skin response (GSR) but did not use these measurements in the analysis since in this, as in our past studies, we have found the GSR to be an unpredictable and unreliable metric [1].
Allowing for a 20% attrition rate – a rate of attrition common in our past studies – we have calculated that we will maintain at least 28 staff and 96 residents per condition at 12-month follow-up.
In our recent study examining the effects of HRLF performance in aged rats [ 19], we observed median neuropathy that was greater than that seen in our past studies on young rats, even studies examining the consequence of a high force task rather than this moderate demand task [ 15, 16, 18, 20].
In the present study, our experiments with subtype-selective antagonists also demonstrated that mERα was the predominant membrane receptor mediating these responses, as we reported previously (Alyea et al. 2008; Jeng and Watson 2011), although, as in our past studies, ERβ and GPR30 also contributed to this ERK response to estrogens.
In our past studies, we used the method published by Horsburgh and co-workers [ 4, 15] that uses a gene replacement vector that features a temperature-sensitive origin of replication (ts-ori), a positive selection marker (zeocin) and negative selection marker (sacB).
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