No standard clinical outcomes are currently established in MCI and pre-MCI populations.
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There is limited evidence of neuropsychological markers in showing the effects of cholinergic precursors in MCI.
Our results provide evidence for the functional disconnection of brain regions in MCI.
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Longitudinal studies assessing PiB binding in MCI reported a cognitive decline along with an increase ligand uptake [43, 44].
Fleisher et al. showed a significant increase of AV-45 uptake in MCI compared to healthy subjects [10].
Information about the nature of functional difficulties in MCI can be used to augment common clinical assessment procedures, and aspects of function that are affected in MCI can serve as meaningful endpoints for intervention trials.
The average total TYM-J scores were 45.7 in controls, 41.7 in MCI, and 35.7 in AD.
We examined whether neuropsychiatric symptoms in MCI increased the risk of incident dementia (all-cause) and incident AD.
To our knowledge no prior study has tested the prognostic value of PMC deactivation in MCI.
