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On the other hand, in both the NE and gentisic septic groups, treatments caused a return in MAP to baseline where MAP remained for the 3 h post- and 5 h post-treatment intervals.
CA was confirmed by an abrupt decrease in MAP to less than 20 mmHg.
The relation between dynamic changes in MAP to CO defines arterial resistance.
ROSC was confirmed by spontaneous cardiac rhythm in conjunction with a rise in MAP to greater than 50 mmHg.
Sensitivity analyses were performed in which a MAP decline of > 10% or an absolute drop in MAP to < 70 mmHg constituted hemodynamic instability.
Compared to baseline, exhaustive exercise significantly suppressed the increase in MAP to visual stimulation, while the CI response did not significantly change by the exercise.
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