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Fig. 6 Oxidative damage products in LLC primary tumor tissue following NP administration to mice.
Templating PNIPAM in LLC mesophases imparts nanostructure to the polymer that significantly increases transport and swelling when compared to isotropic hydrogels.
In order to understand the unique polymerization behavior controlling structural development in LLC media, the polymerization kinetics in these systems have been studied using several commercially available photoinitiators.
The administration of NPs with well-defined topology of metal components (either AgcoreAushell or AucoreAgshell) was the most sufficient in inducing protein carbonylation in LLC primary tumor tissue.
It is possible that WSX1 expression in LLC failed to induce NKG2D ligands.
As expected, WSX1 does not induce NKG2D ligand upregulation in LLC tumor cells where WSX1 promotes tumor growth (Figure 3a).
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Cyclic AMP reverses radiocontrast media induced apoptosis in LLC-PK1 cells by activating A kinase/PI3 kinase.
Transfection of the dominant-negative Akt plasmid resulted in internalization of a part of BCRP in LLC-PK1 cells.
The rescued virus was plaque purified in LLC-MK2 cells, and the stock virus was grown in LLC-MK2 cells.
BSA treatment in LLC-PK1 was performed in cells cultured overnight in medium depleted of serum.
Effect of nocodazole on virus production was also determined in LLC-MK2 cells.
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